锌是称得上的抗空气被氧化剂与抗感染剂  ，在身体里动态平衡、免疫系统基本功能、空气被氧化应激状态、细胞核凋亡和皮肤衰老中起着至关很重要的效果 。锌是人身体必不可少的氢化物发生器营养元素中的一个  ，其是 BB贝博艾弗森官方网站身体300几种酶的辅酶  ，通过设备DNA、RNA 及血清质的炼制  ，可直接影晌生态学膜的可靠性和多血清混合物的布置  ，调试雌激素试述感觉的养成  ，当机器缺锌  ，不直接影晌免疫系统的情形  ，多氧化物应激状态   ，必然会促使发炎的发生 。 锌也就能够直接影晌无数发炎神经元系数的存在和表现传达着  ，在休外可激发双核神经元对外皮神经元的黏附  ，并对促炎神经元系数的存在兼具危害直接影晌 。缺锌会影响对脂多糖的造成 过度发炎想法  ，较轻多微生态学脓毒血症小鼠的浑身发炎想法和脓毒血症引发的器管软机构损伤易情绪化  ，多肺机构中炎性神经元系数传达  ，这一种炎性神经元系数的传达技术水平或与血清锌浓度值出现负相关的动向 。锌没有现在促使炎性神经元系数分泌液暴增之内  ，还要加片中性粒神经元迁出及上浮趋化系数传达 。从某些相拟的探索结局也就能够得知  ，缺锌使宿主上皮细胞各个促炎神经元系数多  ，较轻发炎想法  ，而获取锌可在必然限度上倒转该工作 。近似于的  ，锌的注入可为显著缓和小鼠急性膀胱发炎想法  ，猪苗胃肠道影响  ，必然限度上可用于抗菌素的安全使用  ，是临床试验抗感染剂的内在的得票数者  ，发展潜力大量 。

锌离子（甘氨酸锌Zinc Glycinate、L-天门冬氨酸锌Zinc Aspartate 、乳清酸锌Zinc Orotate、抗坏血酸锌Zinc Ascorbate、葡萄糖酸锌Zinc Gluconate、柠檬酸锌Zinc Citrate、苹果酸锌Zinc Malate、吡啶甲酸锌Zinc Picolinate）的免疫抑制的作用措施

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甘氨酸钙Calcium Glycinate、巨峰葡糖酸锰Manganese Gluconate、富马酸亚铁Ferrous Fumarate、天门冬氨酸镁Magnesium Aspartate、碳水化合物螯合铜Copper Amino Acid Chelate、赖氨酸镁Magnesium Lysinate、L-酪氨酸L-Tyrosine、L-半胱氨酸L-Cysteine、L-鸟氨酸盐酸盐L-Ornithine Hydrochloride、苹果6手机酸钙Calcium Malate

Anti-inflammatory mechanism of zinc ion（Zinc Glycinate、Zinc Aspartate 、Zinc Orotate、Zinc Ascorbate、Zinc Gluconate、Zinc Citrate、Zinc Malate、Zinc Picolinate）

Zinc is a recognized antioxidant and anti-inflammatory agent that plays a vital role in homeostasis, immune function, oxidative stress, apoptosis and aging. Zinc is one of the essential trace elements in the human body. As a coenzyme of more than 300 enzymes in the body, it is involved in the synthesis of DNA, RNA and protein in the body, which can affect the stability of biofilm and the arrangement of multi-protein complexes, and regulate the formation of hormones and their receptors. When the body is deficient in zinc, it will not only affect the immune state, increase oxidative stress, but also lead to inflammation. Zinc can affect the production and signaling of many inflammatory cytokines, enhance the adhesion of monocytes to endothelial cells in vitro, and adversely affect the production of pro-inflammatory cytokines. Zinc deficiency can cause excessive inflammatory response to lipopolysaccharide, aggravate systemic inflammatory response and susceptibility to sepsis induced organ damage in mice with multimicrobial sepsis, and increase the expression of inflammatory cytokines in lung tissue, which may be negatively correlated with serum zinc concentration. In addition to the increased secretion of inflammatory cytokines, zinc deficiency can also increase neutrophil migration and up-regulate the expression of chemokines. From these similar results, it can be seen that zinc deficiency increases various proinflammatory cytokines in the host and aggravates the inflammatory response, and zinc supplementation can reverse this process to a certain extent. Similarly, the addition of zinc can significantly improve the acute inflammatory response of mice and intestinal infection of piglets, and can replace the use of antibiotics to a certain extent, which is a potential candidate for clinical anti-inflammatory agents with broad prospects.

Anti-inflammatory mechanism of zinc ion（Zinc Glycinate、Zinc Aspartate 、Zinc Orotate、Zinc Ascorbate、Zinc Gluconate、Zinc Citrate、Zinc Malate、Zinc Picolinate）

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Calcium Glycinate；Manganese Gluconate；Ferrous Fumarate；Magnesium Aspartate；Copper Amino Acid Chelate；Magnesium Lysinate；L-Tyrosine；L-Cysteine；L-Ornithine Hydrochloride；Calcium Malate